Citation: | YAN Jingxiu, ZHAO Lidong. Effects of thalidomide and baicalein on the proliferation, apoptosis and expression of CRBN protein in HEL cells[J]. Journal of Xuzhou Medical University, 2017, 37(9): 566-570. |
[1] |
Tefferi A. Myelofibrosis with myeloid metaplasia [J].N Engl J Med, 2000,342(17):1255-1265.
|
[2] |
Ribeiro J, Crispim ML, Vagnaldo FF, et al. Potential inhibitory effect of LASSBio-596, a new thalidomide hybrid, on inflammatory corneal angiogenesis in rabbits [J]. Ophthalmic Res, 2012,48(4):177-185.
|
[3] |
Piura B, Medina L, Rabinovich A, et al. Thalidomide distinctly affected TNF-α, IL-6 and MMP secretion by an ovarian cancer cell line (SKOV-3) and primary ovarian cancer cells [J].Eur Cytokine Netw, 2013,24(3):122-129.
|
[4] |
[4 ]Elliott MA, Mesa RA, Li CY,et al. Thalidomide treatment in myelofibrosis with myeloid metaplasia [J]. Br J Haematol, 2002,117(2): 288-296.
|
[5] |
Tefferi A, Cortes J, Verstovsek S, et al.Lenalidomide therapy in myelofibrosis with myeloid metaplasia [J]. Blood, 2006,108(4):1158-1164.
|
[6] |
Begna KH, Pardanani A, Mesa R, et al. Long-term outcome of pomalidomide therapy in myelofibrosis [J]. Am J Hematol, 2012,87(1):66-68.
|
[7] |
Broyl A, Kuiper R, van Duin M, et al.High cereblon expression is associated with better survival in patients with newly diagnosed multiple myeloma treated with thalidomide maintenance [J]. Blood, 2013,121(4):624-627.
|
[8] |
Zhu YX, Braggio E, Shi CX, et al.Cereblon expression is required for the antimyeloma activity of lenalidomide and pomalidomide[J]. Blood, 2011, 118(18): 4771-4779.
|
[9] |
张锐波,何莉,黄赞,等.黄芩素与来那度胺联合诱导骨髓瘤细胞凋亡的协同作用及其机制研究[J].中华血液学杂志,2013,34(6):546-547.
|
[10] |
Sderstrm M, Bolling A, Hammarstrm S, et al.Induction of leukotriene C4 synthase activity in differentiating human erythroleukemia cells[J].Biochem Biophys Res Commun, 1992, 189(2): 1043-1049.
|
[11] |
Accardi A, Picollo A.CLC channels and transporters: proteins with borderline personalities [J]. Biochim Biophys Acta, 2010, 1798(8): 1457-1464.
|
[12] |
Ito T, Ando H, Suzuki T, et al.Identification of a primary target of thalidomide teratogenicity[J]. Science, 2010, 327 (5971): 1345-1350.
|
[13] |
Lu G, Middleton RE, Sun HH, et al.The myeloma drug lenalidomide promotes the cereblon-dependent destruction of Ikaros proteins[J]. Science, 2014,343(6168): 305-309.
|
[14] |
Krnke J, Udeshi ND, Narla A,et al. Lenalidomide causes selective degradation of IKZF1 and IKZF3 in multiple myelomacells[J]. Science, 2014, 343(6168): 301-305.
|
[15] |
Fang J, Liu XN, Bolanos L,et al.A calcium- and calpain-dependent pathway determines the response to lenalidomide in myelodysplastic syndromes[J]. Nat Med, 2016, 22(7): 727-734.
|
[16] |
Hasselbalch HC. Chronic inflammation as a promotor of mutagenesis in essential thrombocythemia, polycythemia vera and myelofibrosis. A human inflammation model for cancer development?[J].Leuk Res, 2013, 37(2): 214-220.
|
[17] |
吴小谢,薛连国,赵利东,等.干扰素联合沙利度胺对HEL细胞凋亡及JAK2V617F突变基因表达的影响[J]. 中国实验血液学杂志,2016, 24(4):998-1002.
|
[18] |
Tefferi A, Lasho TL, Mesa RA,et al. Lenalidomide therapy in del (5) (q31)-associated myelofibrosis: cytogenetic and JAK2V617F molecular remissions[J]. Leukemia, 2007, 21(8): 1827-1828.
|
[19] |
周凝溪. Cereblon在慢性骨髓增殖性肿瘤中表达的临床意义[D].石家庄:河北医科大学,2015.
|